Causative agent
Creutzfeldt-Jakob disease (CJD) is a rare disease that causes the brain to degenerate and become spongy, leading to dementia and death. It belongs to a group of diseases called Transmissible Spongiform Encephalopathies (TSEs) that affect humans and other animals. TSEs are thought to be caused by the buildup of an abnormal, transmissible protein called 'prion' in the brain.
CJD can be grouped under four categories according to their aetiologies: sporadic (about 85% of all CJD cases), familial (5 - 15%), iatrogenic and variant.
Clinical features
The clinical feature of CJD is characterised by rapidly progressive dementia associated with multifocal neurological signs. Early symptoms include memory loss, unsteady gait and loss of coordination of limbs. These dementia-like symptoms will worsen and twitching of limbs and trunk will occur. Besides, visual disturbance, abnormal behaviours and seizures can occur. Most patients die within 1 year after onset of symptoms.
Mode of transmission
The mode of transmission depends on the category of CJD:
Incubation period
Incubation periods for CJD are typically long, measured in terms of years and may exceed 50 years.
Management
Currently, there is no effective treatment or cure for any forms of CJD. Management remains supportive. No specific therapy has been shown to stop the progression of these diseases.
Prevention
Based on the understanding that vCJD is linked to the consumption of bovine products contaminated with the BSE agent, the World Health Organization recommended that countries should not permit tissues that are likely to contain the BSE agent to enter any food chain.